PathMap Literature Based Discovery Engine
Share your Discovery:
Guaranteed to find unpublished literature based discoveries.
Why are frontotemporal dementia and c9orf72 ALS considered different diseases if they are both driven by the same abnormal expansion of a GGGGCC (G₄C₂) sequence in the first intron of the C9orf72 gene? Is it logical to think that CRISPR therapeutics for FTD potentially be used for ALS as well?
C9orf72 expansion
_gates_from_c9orf72_expansion
Gene Expression Regulation
_gates_to_gene_expression_regulation
_gates_from_gene_expression_regulation
Protein Aggregation, Pathological
_gates_to_protein_aggregation__pathological
_gates_from_protein_aggregation__pathological
CRISPR-Cas Systems
_gates_to_crispr_cas_systems
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