PathMap Literature Based Discovery Engine

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Experiment #00000083

Dietary strategy: High amylose maize starch may be identified as a non-invasive tool to improve outcomes in TBI, and potentially hypoxic neurovascular damage, suggesting it could be repurposed for high-altitude workers or elderly patients with cognitive frailty.

High Amylose Maize Starch (HAMS) _gates_from_high_amylose_maize_starch__hams_ Fatty Acids, Volatile _gates_to_fatty_acids__volatile _gates_from_fatty_acids__volatile Blood-Brain Barrier _gates_to_blood_brain_barrier _gates_from_blood_brain_barrier Brain Injuries, Traumatic _gates_to_brain_injuries__traumatic
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Experiment #00000077

The use of lysosome-acidifying nanoparticles (AcNPs) could potentially be adapted to treat nanoplastic-induced lysosomal alkalization or membrane rigidification, as these nanoparticles act to restore the degradative capacity lost during environmental contaminant stress.

Nanoplastic exposure _gates_from_nanoplastic_exposure Lysosomes _gates_to_lysosomes _gates_from_lysosomes Autophagy _gates_to_autophagy Nanoparticles _gates_from_nanoparticles Hydrogen-Ion Concentration
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Experiment #00000004

Can the identified lysosomal-targeted acidic nanoparticles (AcNPs) or specific small-molecule chaperones effectively reverse established alpha-synuclein aggregation in chronic exposure models, and do they provide neuroprotection against subsequent environmental pollutant insults?

Lysosomal Storage Diseases _gates_from_lysosomal_storage_diseases alpha-Synuclein _gates_to_alpha_synuclein Alpha-synuclein aggregation _gates_from_alpha_synuclein_aggregation Molecular Chaperones _gates_to_molecular_chaperones Lysosomes _gates_from_lysosomes
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